Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein | Journal of Neuroscience
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Defining the Neuropathological Aggresome across in Silico, in Vitro, and ex Vivo Experiments | The Journal of Physical Chemistry B
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Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer's disease | SpringerLink
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Ablation of Cellular Prion Protein Does Not Ameliorate Abnormal Neural Network Activity or Cognitive Dysfunction in the J20 Line of Human Amyloid Precursor Protein Transgenic Mice | Journal of Neuroscience
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